To help minimize the damage of acid reflux on your teeth, try to avoid foods that trigger heartburn and acid reflux, such as the following:. In consultation with your doctor, add the following foods to your diet to help reduce the occurrence of heartburn and acid reflux:. Vegetables green beans, broccoli, asparagus, cauliflower, leafy greens, potatoes and cucumbers.
Lean meats chicken, turkey, fish and seafood grilled, broiled baked or poached — avoid fried meat. Healthy fats avocados, walnuts, flaxseed, olive oil, sesame oil and sunflower oil. Links to external sites are provided for your convenience in locating related information and services.
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Policy limitations and exclusions apply. In the event of a conflict between this document and the language stated in your Guardian insurance policy, the language of the policy shall control. The dentist can recommend various treatments such as veneers that can improve the look of your mouth and smile. Teach your child to spit rather than swallow the toothpaste.
Avoid fizzy soft drinks, which are highly acidic and can erode tooth enamel. Drink fluoridated tap water instead. Cut back on sweet or sticky foods such as biscuits or lollies. Chew sugar-free gum to encourage a steady flow of saliva.
Pay careful attention to your tooth brushing and flossing habits. Clean your teeth thoroughly at least twice a day. If your medication is acidic, rinse your mouth with water after taking it and avoid brushing your teeth for at least 30 minutes. Visit your dentist at least once or twice a year. Ask your doctor, dentist and pharmacist about the medicines you take and if they may affect your dental health.
Minimise your intake of alcohol. Consider quitting smoking. If you have a drug dependence problem, consider talking to your doctor about entering a drug treatment program. Your doctor and dentist may offer further self-care suggestions. Follow these suggestions carefully. Medications and dental treatment If you are scheduled for dental treatment, tell your dentist about any medications you are taking or have recently taken, including prescription, over-the-counter and herbal medications.
For more information about public dental services, Tel. Give feedback about this page. Was this page helpful? Yes No. They are characterized by acanthosis, basal cell degeneration, hyperparakeratosis, numerous chronic inflammatory cell infiltrate throughout the connective tissue especially the plasma cells and histiocytes.
The results of this study showed 21 patients with lichenoid reactions. The results were similar to the studies of Mandys et al ,[ 9 ] but this study results did not correlate with the studies of Christensen et al [ 8 ] as their investigation did not support the hypothesis of relationship between lichen planus and hypertension.
Facial nerve paralysis in hypertension is because of edema or hemorrhage in the facial canal, but the exact etiology is unknown. Usually facial nerve paralysis is seen in patients with malignant hypertension. But it is more significant in patients with increased diastolic blood pressure rather than increase in systolic blood pressure. Facial nerve paralysis was assessed clinically by paralysis of facial musculature, drooling of saliva from the corner of the mouth, loss of wrinkles on the forehead with expressionless appearance, inability to blow, inability to close the eyes and continuous rolling of tears.
The results of this present study were similar to the study results of Margabanthu et al , Ellis et al. Gingival enlargement is also one of the most common clinical finding in patients with ypertension taking anti-hypertensive medication especially calcium channel blockers. Gingival enlargements appear clinically as firm nodules of gingival overgrowth seen on either buccal or facial aspects and lingual or palatal aspects of the marginal gingiva.
Sometimes they may even the entire crown causing difficulty in eating. The drugs, which cause the gingival enlargement are amlodipine, nifedipine. The results of this study showed that 79 patients with gingival enlargement who were under either amlodipine or nifedipine therapy.
These results were not significant to the studies of Lawrence et al ,[ 11 ] and Lafzi et al and Barclay et al. But the finding failed to assess whether the periodontitis is due to the hypertension per se or not. The mucosal changes were mainly due to the anti-hypertensive drugs. There was some group of people showing increased incidence of dental caries, this may be correlated with the hypo salivation in patients who were under anti-hypertensive therapy.
Facial nerve paralysis was associated with the malignant hypertension. This study concludes the need to do further studies on the relation between hypertension and periodontitis, the relation between hypertension and facial nerve paralysis.
An affirmative response to at least one of the five following questions about symptoms was shown to correlate with a decrease in saliva:. Xerostomia, also known as dry mouth, is marked by a significant reduction in the secretion of saliva.
Erythema multiforme is a skin condition of unknown cause, possibly mediated by deposition of immune complex mostly IgM in the superficial microvasculature of the skin and oral mucous membrane that usually follows an infection or drug exposure. It is a common disorder, with peak incidence in the second and third decades of life.
AIDS A severe immunological disorder caused by the retrovirus HIV, resulting in a defect in cell-mediated immune response that is manifested by increased susceptibility to opportunistic infections and to certain rare cancers, especially Kaposi's sarcoma. It is transmitted primarily by exposure to contaminated body fluids, especially blood and semen.
Multiple sclerosis A chronic autoimmune disease of the central nervous system in which gradual destruction of myelin occursin patches throughout the brain or spinal cord or both, interfering with the nerve pathways and causing muscular weakness, loss of coordination, and speech and visual disturbances.
Facial nerve paralysis is a common problem that involves the paralysis of any structures innervated by the facial nerve. The pathway of the facial nerve is long and relatively convoluted, and so there are a number of causes that may result in facial nerve paralysis. Lichen planus A dermatologic disease of unknown etiology that also occurs in the mouth, on the tongue, or on the lips as smooth lacy networks of white lines or, less commonly, as white patches that may become ulcerative.
Acanthosis Any thickening of the prickle-cell layer of the epidermis; associated with many skin diseases.
Source of Support: Nil. Conflict of Interest: None declared. National Center for Biotechnology Information , U. J Oral Maxillofac Pathol. Low-pH mouth rinses: Some of the proprietary mouth rinses available for purchase by the general public are shown to be acidic. Salivary flow stimulants and artificial saliva with low pH and high titratable acidity can lead to dental hard tissue demineralization, especially in patients with reduced salivary protection.
Bleaching agents: Some bleaching agents available in the market have an acidic pH. This is mainly to avoid their degradation and thereby increase storage time. It was observed in an in vitro study that acidic bleaching agents resulted in significantly higher enamel hardness loss when compared to less acidic agents. The medications that have the potential to cause erosion of the dental hard tissue secondary to their side effects are mentioned below:.
Drug-induced hyposalivation: Saliva plays an essential role in preserving the surface integrity of dental hard tissues. The protective role of saliva against dental erosion can be attributed to the following factors: 34, Therefore, the medications that cause reduced salivary flow can put the patient at risk of tooth erosion by reducing the protective function of saliva against extrinsic as well as intrinsic acids Figure 4. Drug-induced gastroesophageal reflux: Drugs likely to cause gastroesophageal reflux disease can cause the intrinsic gastric acid to reach the oral cavity and thus increase the risk for dental erosion.
For more information, refer to the article by Bartlett and Smith. Drug-induced vomiting: Drugs that induce vomiting can also be considered an indirect cause of dental erosion. For example, abuse of ipecac syrup an over-the-counter emetic by bulimics can result in dental erosion. Similarly, patients undergoing cytotoxic chemotherapeutic drug treatment for malignancies may suffer from frequent vomiting, resulting in erosion.
Thus, extended use of such drugs can cause dental erosion as a secondary side effect. Various measures to prevent and reduce the incidence of dental erosion related to medications have been summarized in Table 1 as The 9 Rs in the management of dental erosion.
Various medications can either directly or indirectly be implicated as causative factors in the etiopathogenesis of tooth erosion. It is the responsibility of dental professionals to educate patients and medical practitioners about the different precautions that can be taken to prevent and control therapeutic medication—related dental erosion. Manuel S. Tooth structure, saliva and critical pH.
Tooth Erosion: Prevention and Treatment. Abrahamsen TC. The worn dentition—pathognomonic patterns of abrasion and erosion. Int Dent J. Shaw L, Smith AJ. Dental erosion—the problem and some practical solutions. Br Dent J. Boland TW. Dental erosion: more acid means fewer teeth. Buonocore memorial lecture. Dental erosion.
Oper Dent. Addy M, Shellis RP. Interaction between attrition, abrasion and erosion in tooth wear. Monogr Oral Sci. Lussi A. Erosive tooth wear — a multifactorial condition of growing concern and increasing knowledge. Bartlett DW.
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